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Pregnancy Timeline by SemestersFemale Reproductive SystemFertilizationThe Appearance of SomitesFirst TrimesterSecond TrimesterThird TrimesterFetal liver is producing blood cellsHead may position into pelvisBrain convolutions beginFull TermWhite fat begins to be madeWhite fat begins to be madeHead may position into pelvisImmune system beginningImmune system beginningPeriod of rapid brain growthBrain convolutions beginLungs begin to produce surfactantSensory brain waves begin to activateSensory brain waves begin to activateInner Ear Bones HardenBone marrow starts making blood cellsBone marrow starts making blood cellsBrown fat surrounds lymphatic systemFetal sexual organs visibleFinger and toe prints appearFinger and toe prints appearHeartbeat can be detectedHeartbeat can be detectedBasic Brain Structure in PlaceThe Appearance of SomitesFirst Detectable Brain WavesA Four Chambered HeartBeginning Cerebral HemispheresEnd of Embryonic PeriodEnd of Embryonic PeriodFirst Thin Layer of Skin AppearsThird TrimesterDevelopmental Timeline
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November 16, 2012--------News Archive Return to: News Alerts


Proteinuria or protein in uria (urine) is a strong indicator for pre-eclampsia.











WHO Child Growth Charts

       

AGT2R Gene Points to Pre-Eclampsia Pregnancy Complication

New research at the University of Adelaide has revealed a genetic link in pregnant moms - and their male partners - to pre-eclampsia, a life-threatening complication during pregnancy

Pre-eclampsia involves high blood pressure and fluid retention and can cause damage to the kidneys and liver. About 7% of pregnancies are affected by pre-eclampsia.


In a paper now online in the journal Placenta ahead
of print publication, the researchers say they have found
a genetic variant involving the AGT2R gene,
which may predispose women to pre-eclampsia.

However, the genetic variant is only associated with
pre-eclampsia when the pregnant mother is
overweight or obese.


"Being able to predict which women are at risk of pre-eclampsia is a very important goal in obstetrics," says Professor Claire Roberts from the University of Adelaide's Robinson Institute.

Professor Roberts, Dr Ang Zhou and Professor Gus Dekker from the Robinson Institute studied data from the SCOPE study, involving more than 2000 women and their partners in Adelaide, Australia and Auckland, New Zealand.


Women who developed pre-eclampsia who were
also overweight or obese were twice as likely to
carry the AGT2R gene variant than the common
form of the gene.

The male partners of women with pre-eclampsia
were also twice as likely to carry the variant gene.

Their babies were three times more
likely to carry the variant.

"This is a condition that can run in families.
With both the mother and the father passing
on their variant genes to their children,
this places the child at greater risk of
parenting a pre-eclamptic pregnancy."


Claire Roberts
University of Adelaide's Robinson Institute


Professor Roberts says the genetic variant is linked with restricted blood flow to the placenta.

"Impaired blood flow in the uterine artery is characterized by a 'notching effect' that appears on a Doppler ultrasound at 20 weeks gestation. Uterine artery notching has previously been associated with pre-eclampsia, and this restricted blood flow is due to impaired placental development," Professor Roberts says.

The researchers say the genetic variant has only a subtle effect in women of normal weight, but in overweight and obese women it appears to independently contribute to the risk of pre-eclampsia.

"Understanding this association could help to predict which women are likely to develop pre-eclampsia," Professor Roberts says.

"However, it also helps to reinforce the message that a normal weight prior to pregnancy will lower the risk of serious complications - being overweight or obese increases the risk of complications."

Original article: http://www.adelaide.edu.au/news/news57761.html