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Welcome to The Visible Embryo, a comprehensive educational resource on human development from conception to birth.

The Visible Embryo provides visual references for changes in fetal development throughout pregnancy and can be navigated via fetal development or maternal changes.

The National Institutes of Child Health and Human Development awarded Phase I and Phase II Small Business Innovative Research Grants to develop The Visible Embryo. Initally designed to evaluate the internet as a teaching tool for first year medical students, The Visible Embryo is linked to over 600 educational institutions and is viewed by more than ' million visitors each month.


WHO International Clinical Trials Registry Platform
The World Health Organization (WHO) has created a new Web site to help researchers, doctors and patients obtain reliable information on high-quality clinical trials. Now you can go to one website and search all registers to identify clinical trial research underway around the world!



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Disclaimer: The Visible Embryo web site is provided for your general information only. The information contained on this site should not be treated as a substitute for medical, legal or other professional advice. Neither is The Visible Embryo responsible or liable for the contents of any websites of third parties which are listed on this site.
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Pregnancy Timeline by SemestersFetal liver is producing blood cellsHead may position into pelvisBrain convolutions beginFull TermWhite fat begins to be madeWhite fat begins to be madeHead may position into pelvisImmune system beginningImmune system beginningPeriod of rapid brain growthBrain convolutions beginLungs begin to produce surfactantSensory brain waves begin to activateSensory brain waves begin to activateInner Ear Bones HardenBone marrow starts making blood cellsBone marrow starts making blood cellsBrown fat surrounds lymphatic systemFetal sexual organs visibleFinger and toe prints appearFinger and toe prints appearHeartbeat can be detectedHeartbeat can be detectedBasic Brain Structure in PlaceThe Appearance of SomitesFirst Detectable Brain WavesA Four Chambered HeartBeginning Cerebral HemispheresFemale Reproductive SystemEnd of Embryonic PeriodEnd of Embryonic PeriodFirst Thin Layer of Skin AppearsThird TrimesterSecond TrimesterFirst TrimesterFertilizationDevelopmental Timeline
Click weeks 0 - 40 and follow fetal growth
Google Search artcles published since 2007
 
August 19, 2011--------News Archive

Hydrodynamics Transform Embryonic Cells Into Us
H
ydrodynamics can contribute to our understanding of how a cluster of embryonic cells can transform into an animal.

New Data on Adenine, a Crucial Building Block of Life
The five nucleic acids making up DNA are some of the few that can withstand ultraviolet light. But adenine turns out to have an extensive range of respones.


August 18, 2011--------News Archive

Pluripotent Stem Cells Developmentally Immature
Researchers have discovered that though similar, induced pluripotent stem cells are similar to embryonic stem cells, but are much more developmentally immature.

Change the Environment, Not the Child
National study finds equal benefit for children with cerebral palsy.


August 17, 2011--------News Archive

Molecular Delivery Serves Gene Therapy Cocktail
Scientists have devised a gene therapy cocktail that has the potential to treat some inherited diseases associated with "misfolded" proteins.

Children of Depressed Mothers Have a Different Brain
MRI scans show their children have an enlarged amygdala.

Discovery Likely to Spur Medicine and Human Health
Scientists have gained new insight into the relationship between two proteins that, out of balance, can prevent normal development of stem cells in the heart.


August 16, 2011--------News Archive

Study Finds New Role for Protein in Hearing
A protein involved in sound sensing in the inner ear may also play a role in transmitting sound information to the brain.

Retinoblastoma Made of Hybrid Cells
Scientists settle a century-old debate about retinoblastoma's beginnings and identify new targets for treating the childhood eye tumor.

Can Oral Care for Babies Prevent Future Cavities?
A recent study confirms the presence of bacteria associated with early childhood caries (ECC) in infant saliva.


August 15, 2011--------News Archive

Slowing the Allergic March
Researchers identify a target that could combat allergies of early childhood.

Gene Clue in the Development of Rheumatoid Arthritis
Findings will help lead to personalized therapies for common, complex illnesses characterized by abnormal immune responses.

Sight Re-Constructs Moving Objects: One by One
Our visual system groups areas of the world with similar characteristics, such as color, shape, or motion.WHO Child Growth Charts

Millions of Canadians are affected by autoimmune disorders that are a common cause of long-term pain and/or disability.

Scientists at Mount Sinai Hospital, in collaboration with researchers at the University of Toronto, University Health Network and McGill University now have significant new insights into the causes of rheumatoid arthritis (RA) and other autoimmune disorders including type 1 diabetes, lupus and Graves disease.

The findings represent a key initial step in realizing the full potential of genomics and personalized medicine.

In a study published online today in Nature Genetics, Dr. Katherine Siminovitch and her team identified the exact means by which an alteration in the gene PTPN22 increases risk for RA and other autoimmune disorders. The study used advanced genomics technologies that enable testing of millions of genetic markers in a single experiment to identify genes, such as PTPN22, that confer risk for disease.

The team then generated a mouse genetic model to show how the PTPN22 gene mutation impairs immune cell function and then validating their findings in humans, taking their discovery from the laboratory bench to the clinic.

The result: a more accurate understanding of how autoimmune conditions develop, and how new diagnostic tests and targeted therapies can be designed for better symptom control and potential cure.

"Our findings are particularly exciting because this study sets a new precedent for studying arthritis and other autoimmune disorders," said lead author Dr. Siminovitch, Senior Investigator and the Sherman Family Research Chair in Genomic Medicine at the Samuel Lunenfeld Research Institute of Mount Sinai Hospital, a professor at the University of Toronto, and Director of the Fred A. Litwin & Family Centre in Genetic Medicine.

"This is one of the first studies in which we have traced the steps that connect a specific genetic lesion to the development of a common, complex autoimmune condition."

Led by Dr. Siminovitch, the group used genetically modified mice in which PTPN22 had been altered to mimic a genetic mutation found in many RA patients. The effects of this change on immune cells were observed in the mice, and the studies were then repeated in human blood samples from patients with and without RA. By this means, the group honed in on the impact of a key protein called Lyp/Pep that—in healthy cells—prevents the hyper-immune responses that lead to autoimmune disorders.

The group found that this gene mutation leads to decreased levels of Lyp, thereby removing a natural brake that normally prevents the inflammatory processes underlying RA and many other autoimmune conditions.

"Measuring levels of this protein will help us monitor disease severity in patients with autoimmune disorders, test the effects of various therapies including new drugs, and determine which treatments work best in specific patients," said Dr. Edward Keystone, co-author of the study and Director of the Rebecca MacDonald Centre for Arthritis and Autoimmune Disease at Mount Sinai Hospital. "We are truly seeing genomics in action with this study, and the results give us new hope for improving patient outcomes."

Dr. Keystone emphasized the importance of this type of research to the practice of medicine in general, noting that advances in genetics knowledge are allowing for earlier diagnoses and more personalized treatments that give patients better outcomes.

"Using the powerful genetic tools now available, previously cryptic diseases are being dissected and their underlying causes identified," said Dr. Jim Woodgett, the Lunenfeld's Director of Research. "Drs. Siminovitch and Keystone are at the leading edge of employing these genomic approaches for the benefit of patients, seamlessly combining their research skills with clinical insights."

The study was funded by Canadian Institutes of Health Research, the Canadian Arthritis Network, and the Ontario Research Fund.

Mount Sinai Hospital is an internationally recognized, 472-bed acute care academic health sciences centre affiliated with the University of Toronto. It is known for excellence in the provision of compassionate patient care, innovative education, and leading-edge research. Mount Sinai's Centres of Excellence include: Daryl A. Katz Centre for Urgent & Critical Care; Lawrence and Frances Bloomberg Centre for Women's & Infants' Health; Christopher Sharp Centre for Surgery & Oncology; Centre for Inflammatory Bowel Disease; Centre for Musculoskeletal Disease and the Samuel Lunenfeld Research Institute. For more information about Mount Sinai Hospital, please visit www.mountsinai.ca.

The Samuel Lunenfeld Research Institute of Mount Sinai Hospital, a University of Toronto affiliated research centre established in 1985, is one of the world's premier centres in biomedical research. Thirty-six principal investigators lead research in diabetes, cancer biology, epidemiology, stem cell research, women's and infants' health, neurobiology and systems biology. For more information on the Samuel Lunenfeld Research Institute, please visit www.lunenfeld.ca.

Original article: http://www.eurekalert.org/pub_releases/2011-08/slri-trf081011.php

IMAGE: High-fat diet and obesity cause beta cells to lose their ability to sense glucose in the blood. Left: Pancreatic beta cells from a mouse on a standard diet. Right: Pancreatic...
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